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human bronchial epithelial cells  (ATCC)


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    ATCC human bronchial epithelial cells
    HDM d oes not have mutagenic effects in human and mouse lung <t>epithelial</t> cell lines i n v itro . A) Dose-response curves of HDM treatment in BEAS-2B and LLC cells. Cell viability is shown relative to vehicle control (PBS; % of control). Calculated IC 50 values are indicated. B) Experimental setup for HDM exposure and duplex sequencing. BEAS-2B and LLC cells were treated with either HDM (200 µg/mL for BEAS-2B; 400 µg/mL for LLC) or vehicle (VEH; PBS), expanded across multiple passages, and subsequently subjected to genomic sequencing. C) Comparison of mutational burden between samples treated with either HDM (purple) or VEH (blue), shown as somatic mutations per megabase (Mb) of coding DNA. D) Mutational spectra of BEAS-2B and LLC cells treated with HDM compared to VEH controls. Mutations are classified into the six base substitution categories and displayed in SBS96 format. The experiments were performed in triplicate (A) or singlicate (B-D).
    Human Bronchial Epithelial Cells, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 4158 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/human bronchial epithelial cells/product/ATCC
    Average 99 stars, based on 4158 article reviews
    human bronchial epithelial cells - by Bioz Stars, 2026-02
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    1) Product Images from "Multi-omics profiling reveals microenvironmental remodeling as a key driver of house dust mite-induced lung cancer progression"

    Article Title: Multi-omics profiling reveals microenvironmental remodeling as a key driver of house dust mite-induced lung cancer progression

    Journal: Neoplasia (New York, N.Y.)

    doi: 10.1016/j.neo.2026.101275

    HDM d oes not have mutagenic effects in human and mouse lung epithelial cell lines i n v itro . A) Dose-response curves of HDM treatment in BEAS-2B and LLC cells. Cell viability is shown relative to vehicle control (PBS; % of control). Calculated IC 50 values are indicated. B) Experimental setup for HDM exposure and duplex sequencing. BEAS-2B and LLC cells were treated with either HDM (200 µg/mL for BEAS-2B; 400 µg/mL for LLC) or vehicle (VEH; PBS), expanded across multiple passages, and subsequently subjected to genomic sequencing. C) Comparison of mutational burden between samples treated with either HDM (purple) or VEH (blue), shown as somatic mutations per megabase (Mb) of coding DNA. D) Mutational spectra of BEAS-2B and LLC cells treated with HDM compared to VEH controls. Mutations are classified into the six base substitution categories and displayed in SBS96 format. The experiments were performed in triplicate (A) or singlicate (B-D).
    Figure Legend Snippet: HDM d oes not have mutagenic effects in human and mouse lung epithelial cell lines i n v itro . A) Dose-response curves of HDM treatment in BEAS-2B and LLC cells. Cell viability is shown relative to vehicle control (PBS; % of control). Calculated IC 50 values are indicated. B) Experimental setup for HDM exposure and duplex sequencing. BEAS-2B and LLC cells were treated with either HDM (200 µg/mL for BEAS-2B; 400 µg/mL for LLC) or vehicle (VEH; PBS), expanded across multiple passages, and subsequently subjected to genomic sequencing. C) Comparison of mutational burden between samples treated with either HDM (purple) or VEH (blue), shown as somatic mutations per megabase (Mb) of coding DNA. D) Mutational spectra of BEAS-2B and LLC cells treated with HDM compared to VEH controls. Mutations are classified into the six base substitution categories and displayed in SBS96 format. The experiments were performed in triplicate (A) or singlicate (B-D).

    Techniques Used: Control, Sequencing, Genomic Sequencing, Comparison



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    HDM d oes not have mutagenic effects in human and mouse lung epithelial cell lines i n v itro . A) Dose-response curves of HDM treatment in BEAS-2B and LLC cells. Cell viability is shown relative to vehicle control (PBS; % of control). Calculated IC 50 values are indicated. B) Experimental setup for HDM exposure and duplex sequencing. BEAS-2B and LLC cells were treated with either HDM (200 µg/mL for BEAS-2B; 400 µg/mL for LLC) or vehicle (VEH; PBS), expanded across multiple passages, and subsequently subjected to genomic sequencing. C) Comparison of mutational burden between samples treated with either HDM (purple) or VEH (blue), shown as somatic mutations per megabase (Mb) of coding DNA. D) Mutational spectra of BEAS-2B and LLC cells treated with HDM compared to VEH controls. Mutations are classified into the six base substitution categories and displayed in SBS96 format. The experiments were performed in triplicate (A) or singlicate (B-D).

    Journal: Neoplasia (New York, N.Y.)

    Article Title: Multi-omics profiling reveals microenvironmental remodeling as a key driver of house dust mite-induced lung cancer progression

    doi: 10.1016/j.neo.2026.101275

    Figure Lengend Snippet: HDM d oes not have mutagenic effects in human and mouse lung epithelial cell lines i n v itro . A) Dose-response curves of HDM treatment in BEAS-2B and LLC cells. Cell viability is shown relative to vehicle control (PBS; % of control). Calculated IC 50 values are indicated. B) Experimental setup for HDM exposure and duplex sequencing. BEAS-2B and LLC cells were treated with either HDM (200 µg/mL for BEAS-2B; 400 µg/mL for LLC) or vehicle (VEH; PBS), expanded across multiple passages, and subsequently subjected to genomic sequencing. C) Comparison of mutational burden between samples treated with either HDM (purple) or VEH (blue), shown as somatic mutations per megabase (Mb) of coding DNA. D) Mutational spectra of BEAS-2B and LLC cells treated with HDM compared to VEH controls. Mutations are classified into the six base substitution categories and displayed in SBS96 format. The experiments were performed in triplicate (A) or singlicate (B-D).

    Article Snippet: Human bronchial epithelial cells (BEAS-2B) were purchased in 2018 from ATCC (Cat. No CRL-9609).

    Techniques: Control, Sequencing, Genomic Sequencing, Comparison